vikas sood's profile
What I do
Student
Affiliations
Current affiliations
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- Position
- Company
- National Institute of Immunology
- Further information
Location
- City:
- None chosen
- Hub:
- None chosen
Interests
Primarily my interest revolves around the molecular biology and Immunology of HIV-1.
Projects
Design of Efficient Synthetic Nucleic Acids Inhibiting HIV-1 Replication.
Role of Host Genes in HIV-1 Progression.
Genetic variations in HIV-1 Subtype C prevalent in India.
Publications
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Risk for HIV-1 infection is not associated with Repeat-Region polymorphism in the DC-SIGN neck domain and Novel Genetic DC-SIGN Variants among North Indians. Clinica chimica acta; international journal of clinical chemistry (2008) (Epub 12 Jan 2008) PubMed ID:(18255039)
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Bano A, Gupta N, Sharma Y, Sood V, Banerjea A. HIV-1 VprB and C RNA cleavage by potent 10-23 DNAzymes that also cause reversal of G2 cell cycle arrest mediated by Vpr genes. Oligonucleotides (4) , 465-72 (2007) PubMed ID:(18078383)
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Bano A, Gupta N, Sood V, Banerjea A. Vpr from HIV-1 subtypes B and C exhibit significant differences in their ability to transactivate LTR-mediated gene expression and also in their ability to promote apoptotic DNA ladder formation. AIDS (London, England) (13) , 1832-4 (2007) PubMed ID:(17690591)
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Gupta N, Sood V, Bano A, Banerjea A. X Protein of hepatitis B virus potently activates HIV-1 subtype C long terminal repeat promoter: implications for faster spread of HIV-1 subtype C. AIDS (London, England) (11) , 1491-2 (2007) PubMed ID:(17589200)
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Sood V, Gupta N, Bano A, Banerjea A. DNA-enzyme-mediated cleavage of human immunodeficiency virus type 1 Gag RNA is significantly augmented by antisense-DNA molecules targeted to hybridize close to the cleavage site. Oligonucleotides (1) , 113-21 (2007) PubMed ID:(17461768)
Contact
- email:
- vikas1101 [ at ] yahoo.com
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