Jeremie Vitte's profile
What I do
R & D
Neurodegenerative Disorders
Cellular and Molecular Biology
Currently studying the physiopathology of Parkinson’s disease:
• Characterization of the cellular and subcellular distribution of LRRK2 protein in normal or pathogenic human brain.
• Characterization of neuronal damage in a α-synuclein mouse model.
• Analysis of dopaminergic neuron degeneration in old parkin knock out mice.
Affiliations
Current affiliations
-
- Position
- Postdoctoral Researcher
- Company
- INSERM U679 (Ex U289), Neurology and Experimental Therapeutics Laboratory, Paris, France
- Duration
- 2008 - Present
- Further information
Past affiliations
-
- Position
- PhD Researcher
- Company
- INSERM U798, Molecular Neurogenetics Laboratory, Paris Area, France
- Duration
- 2002 - 2007
Location
- City:
- None chosen
- Hub:
- None chosen
Interests
R & D
Neurodegenerative Disorders
Cellular and Molecular Biology
Main skills:
- Generation and characterization of mouse and cellular models
- Study of links between human pathologies and experimental models
- Project management
Projects
Career objective: Understand molecular mechanisms and participate to the development of therapies in human neurodegenerative disorders.
Publications
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Vitte J, Fassier C, Tiziano F, Dalard C, Soave S, Roblot N, Brahe C, Saugier-Veber P, Bonnefont J, Melki J. Refined characterization of the expression and stability of the SMN gene products. The American journal of pathology (4) , 1269-80 (2007) (Epub 23 Aug 2007) PubMed ID:(17717146)
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Tarrade A, Fassier C, Courageot S, Charvin D, Vitte J, Peris L, Thorel A, Mouisel E, Fonknechten N, Roblot N, Seilhean D, Diérich A, Hauw J, Melki J. A mutation of spastin is responsible for swellings and impairment of transport in a region of axon characterized by changes in microtubule composition. Human molecular genetics (24) , 3544-58 (2006) (Epub 13 Nov 2006) PubMed ID:(17101632)
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Vitte J, Davoult B, Roblot N, Mayer M, Joshi V, Courageot S, Tronche F, Vadrot J, Moreau M, Kemeny F, Melki J. Deletion of murine Smn exon 7 directed to liver leads to severe defect of liver development associated with iron overload. The American journal of pathology (5) , 1731-41 (2004) PubMed ID:(15509541)
Jeremie Vitte's activity on Nature Network
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